Mucociliary dysfunction, chronic infections with advancing immunosenescence and pulmonary emphysema as seen in COPD may be just as relevant to ACOS. Possible drug targets include receptors of the ...
After a latent period of 48 to 72 hours, toxin-mediated damage to the bronchial mucosa causes loss of mucociliary transport, bronchial edema, and cellular necrosis. The clinical picture includes ...